Download Adrenal Imaging by Shamlal Mangray, Ronald A. DeLellis (auth.), Michael A. PDF

By Shamlal Mangray, Ronald A. DeLellis (auth.), Michael A. Blake, Giles Boland (eds.)

ISBN-10: 193411586X

ISBN-13: 9781934115862

Imaging of the adrenal gland has made great development within the final decade as new applied sciences proceed to conform. Adrenal Imaging highlights the pertinent scientific and pathological details that underpins the actual interpretation and use of adrenal imaging. Written by way of a prestigious crew of foreign individuals, person chapters in Adrenal Imaging function a suitable and updated reference of adrenal imaging findings, algorithms and strategies in CT, MR nuclear medication, intervention, and trauma. precis sections on the finish of every bankruptcy remove darkness from key instructing issues to augment retention.

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Extra resources for Adrenal Imaging

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Rarely, adrenal medullary hyperplasia may occur in patients without a known hereditary syndrome. The tumors are composed of intermediate to large polygonal cells that may be arranged in alveolar, trabecular, or solid patterns. Most pheochromocytomas exhibit admixtures of these growth patterns. In tumors with alveolar arrangements, the groups of 20 S. A. DeLellis Fig. 11 Gross photograph of pheochromocytoma (A). The characteristic tan cut surface of this tumor contrasts with the yellow rim of adjacent cortex.

Very rare examples of primary lymphomas of the adrenal glands have also been reported [76, 77]. Virtually all of the primary adrenal lymphomas have been of the non-Hodgkin’s large-cell type, including the large-cell angiotropic variant [78]. Cysts and Pseudocysts Although adrenal cysts are uncommon, their rate of detection as a result of CT and MRI scanning has increased dramatically. Most adrenal cysts are unilateral, with a predominance in women. They have been divided into four major categories, including epithelial cysts (retention cysts, embryonal cysts, and cystic neoplasms), parasitic cysts (predominantly echinococcal), endothelial cysts, and pseudocysts [7, 12, 17].

Endocrinologist 2:6–21 28. Groussin L, Jullian E, Perlemoine K et al. (2002) Mutations of the PRKAR1A gene in Cushing’s syndrome due to sporadic primary pigmented nodular adrenal cortical disease. J Clin Endocrinol Metab 87:4324–4329 29. Stratakis CA (2002) Mutations of the gene encoding the protein kinase A Type Ia regulatory subunit (PRKAR1A) in patients with the ‘‘complex of spotty skin pigmentation, myxomas, endocrine overactivity and Schwannomas’’ (Carney Complex) Ann NY Acad Sci 968:3–21 30.

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